Inhibition of Na(V)1.8 prevents atrial arrhythmogenesis in human and mice

Pharmacologic approaches for the treatment of atrial arrhythmias are limited due to side effects and low efficacy. Thus, the identification of new antiarrhythmic targets is of clinical interest. Recent genome studies suggested an involvement of SCN10A sodium channels (Na(V)1.8) in atrial electrophys...

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Detalhes bibliográficos
Publicado no:Basic Res Cardiol
Main Authors: Pabel, Steffen, Ahmad, Shakil, Tirilomis, Petros, Stehle, Thea, Mustroph, Julian, Knierim, Maria, Dybkova, Nataliya, Bengel, Philipp, Holzamer, Andreas, Hilker, Michael, Streckfuss-Bömeke, Katrin, Hasenfuss, Gerd, Maier, Lars S., Sossalla, Samuel
Formato: Artigo
Idioma:Inglês
Publicado em: Springer Berlin Heidelberg 2020
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Original Contribution
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7033079/
https://ncbi.nlm.nih.gov/pubmed/32078054
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00395-020-0780-8
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