Inhibition of Na(V)1.8 prevents atrial arrhythmogenesis in human and mice

Pharmacologic approaches for the treatment of atrial arrhythmias are limited due to side effects and low efficacy. Thus, the identification of new antiarrhythmic targets is of clinical interest. Recent genome studies suggested an involvement of SCN10A sodium channels (Na(V)1.8) in atrial electrophys...

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Bibliografiske detaljer
Udgivet i:Basic Res Cardiol
Main Authors: Pabel, Steffen, Ahmad, Shakil, Tirilomis, Petros, Stehle, Thea, Mustroph, Julian, Knierim, Maria, Dybkova, Nataliya, Bengel, Philipp, Holzamer, Andreas, Hilker, Michael, Streckfuss-Bömeke, Katrin, Hasenfuss, Gerd, Maier, Lars S., Sossalla, Samuel
Format: Artigo
Sprog:Inglês
Udgivet: Springer Berlin Heidelberg 2020
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Original Contribution
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7033079/
https://ncbi.nlm.nih.gov/pubmed/32078054
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00395-020-0780-8
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