Aspirin Enhances the Protection of Hsp90 from Heat-Stressed Injury in Cardiac Microvascular Endothelial Cells Through PI3K-Akt and PKM2 Pathways

Heat stress (HS) often causes sudden death of humans and animals due to heart failure, mainly resulting from the contraction of cardiac microvasculature followed by myocardial ischemia. Cardiac microvascular endothelial cells (CMVECs) play an important role in maintaining vasodilatation. Aspirin (AS...

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Pubblicato in:Cells
Autori principali: Zhang, Xiaohui, Chen, Bixia, Wu, Jiaxin, Sha, Junzhou, Yang, Bo, Zhu, Jie, Sun, Jiarui, Hartung, Jörg, Bao, Endong
Natura: Artigo
Lingua:Inglês
Pubblicazione: MDPI 2020
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Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC7016979/
https://ncbi.nlm.nih.gov/pubmed/31963688
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/cells9010243
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