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Metabolic reprogramming of fibro/adipogenic progenitors facilitates muscle regeneration
In Duchenne muscular dystrophy (DMD), the absence of the dystrophin protein causes a variety of poorly understood secondary effects. Notably, muscle fibers of dystrophic individuals are characterized by mitochondrial dysfunctions, as revealed by a reduced ATP production rate and by defective oxidati...
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| Yayımlandı: | Life Sci Alliance |
|---|---|
| Asıl Yazarlar: | , , , , , , , , , , , , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Life Science Alliance LLC
2020
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7003708/ https://ncbi.nlm.nih.gov/pubmed/32019766 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.26508/lsa.202000660 |
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