Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components

Lignende værker

• Inhibition of the sarco/endoplasmic reticulum (ER) Ca(2+)-ATPase by thapsigargin analogs induces cell death via ER Ca(2+) depletion and the unfolded protein response
  af: Sehgal, Pankaj, et al.
  Udgivet: (2017)
• Targeting thapsigargin towards tumors
  af: Doan, Nhu Thi Quynh, et al.
  Udgivet: (2014)
• Critical Roles of Hydrophobicity and Orientation of Side Chains for Inactivation of Sarcoplasmic Reticulum Ca(2+)-ATPase with Thapsigargin and Thapsigargin Analogs
  af: Winther, Anne-Marie L., et al.
  Udgivet: (2010)
• Preparation of Enzyme-Activated Thapsigargin Prodrugs by Solid-Phase Synthesis
  af: Zimmermann, Tomas, et al.
  Udgivet: (2018)
• Structure/activity relationship of thapsigargin inhibition on the purified Golgi/secretory pathway Ca(2+)/Mn(2+)-transport ATPase (SPCA1a)
  af: Chen, Jialin, et al.
  Udgivet: (2017)