High glucose reduces expression of podocin in cultured human podocytes by stimulating TRPC6

The transient receptor potential canonical 6 (TRPC6) channel and podocin are colocalized in the glomerular slit diaphragm as an important complex to maintain podocyte function. Gain of TRPC6 function and loss of podocin function induce podocyte injury. We have previously shown that high glucose indu...

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Detalhes bibliográficos
Publicado no:Am J Physiol Renal Physiol
Main Authors: Lu, Xiao-Yu, Liu, Bing-Chen, Cao, Yu-Ze, Song, Chang, Su, Hua, Chen, Guangping, Klein, Janet D., Zhang, Hui-Xue, Wang, Li-Hua, Ma, He-Ping
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2019
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6960785/
https://ncbi.nlm.nih.gov/pubmed/31566428
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00215.2019
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