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High glucose reduces expression of podocin in cultured human podocytes by stimulating TRPC6

The transient receptor potential canonical 6 (TRPC6) channel and podocin are colocalized in the glomerular slit diaphragm as an important complex to maintain podocyte function. Gain of TRPC6 function and loss of podocin function induce podocyte injury. We have previously shown that high glucose indu...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
में प्रकाशित:Am J Physiol Renal Physiol
मुख्य लेखकों: Lu, Xiao-Yu, Liu, Bing-Chen, Cao, Yu-Ze, Song, Chang, Su, Hua, Chen, Guangping, Klein, Janet D., Zhang, Hui-Xue, Wang, Li-Hua, Ma, He-Ping
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: American Physiological Society 2019
विषय:
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC6960785/
https://ncbi.nlm.nih.gov/pubmed/31566428
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00215.2019
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