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Passive immunotherapies targeting amyloid beta and tau oligomers in Alzheimer’s disease
Alzheimer’s disease (AD) is historically difficult to treat, in part due the inaccessible nature of brain pathology. Amyloid-β (Aβ) and tau proteins drive pathology by forming toxic oligomers that eventually deposit as insoluble amyloid plaques and neurofibrillary tangles. Recent clinical studies su...
Guardat en:
| Publicat a: | J Pharm Sci |
|---|---|
| Autors principals: | , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2019
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6937375/ https://ncbi.nlm.nih.gov/pubmed/31647950 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.xphs.2019.10.024 |
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