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Tight junction protein occludin regulates progenitor Self-Renewal and survival in developing cortex
Occludin (OCLN) mutations cause human microcephaly and cortical malformation. A tight junction component thought absent in neuroepithelium after neural tube closure, OCLN isoform-specific expression extends into corticogenesis. Full-length and truncated isoforms localize to neuroprogenitor centrosom...
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| Pubblicato in: | eLife |
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| Autori principali: | , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
eLife Sciences Publications, Ltd
2019
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6890460/ https://ncbi.nlm.nih.gov/pubmed/31794381 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.49376 |
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