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Proinflammatory NFkB signalling promotes mitochondrial dysfunction in skeletal muscle in response to cellular fuel overloading

Sustained nutrient (fuel) excess, as occurs during obesity and diabetes, has been linked to increased inflammation, impaired mitochondrial homeostasis, lipotoxicity, and insulin resistance in skeletal muscle. Precisely how mitochondrial dysfunction is initiated and whether it contributes to insulin...

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Detalhes bibliográficos
Publicado no:Cell Mol Life Sci
Main Authors: Nisr, Raid B., Shah, Dinesh S., Ganley, Ian G., Hundal, Harinder S.
Formato: Artigo
Idioma:Inglês
Publicado em: Springer International Publishing 2019
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6881256/
https://ncbi.nlm.nih.gov/pubmed/31101940
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00018-019-03148-8
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