An N-/L-type calcium channel blocker, cilnidipine, suppresses autonomic, electrical, and structural remodelling associated with atrial fibrillation

AIMS: Autonomic dysfunction can promote atrial fibrillation (AF) and results from AF-related remodelling. N-type Ca(2+)-channels (NTCCs) at sympathetic nerve terminals mediate Ca(2+)-entry that triggers neurotransmitter release. AF-associated remodelling plays an important role in AF pathophysiology...

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Detaylı Bibliyografya
Yayımlandı:Cardiovasc Res
Asıl Yazarlar: Tajiri, Kazuko, Guichard, Jean-Baptiste, Qi, Xiaoyan, Xiong, Feng, Naud, Patrice, Tardif, Jean-Claude, Costa, Antoine Da, Aonuma, Kazutaka, Nattel, Stanley
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Oxford University Press 2019
Konular:
Original Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6873085/
https://ncbi.nlm.nih.gov/pubmed/31119260
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvz136
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