Maf1 ameliorates cardiac hypertrophy by inhibiting RNA polymerase III through ERK1/2

Rationale: An imbalance between protein synthesis and degradation is one of the mechanisms of cardiac hypertrophy. Increased transcription in cardiomyocytes can lead to excessive protein synthesis and cardiac hypertrophy. Maf1 is an RNA polymerase III (RNA pol III) inhibitor that plays a pivotal rol...

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Pubblicato in:Theranostics
Autori principali: Sun, Yu, Chen, Cong, Xue, Ruicong, Wang, Yan, Dong, Bin, Li, Jiayong, Chen, Chen, Jiang, Jingzhou, Fan, Wendong, Liang, Zhuomin, Huang, Huiling, Fang, Rong, Dai, Gang, Yan, Youchen, Yang, Tiqun, Li, Xiangxue, Huang, Zhan-Peng, Dong, Yugang, Liu, Chen
Natura: Artigo
Lingua:Inglês
Pubblicazione: Ivyspring International Publisher 2019
Soggetti:
Research Paper
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6831308/
https://ncbi.nlm.nih.gov/pubmed/31695767
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7150/thno.33006
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