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Maf1 ameliorates cardiac hypertrophy by inhibiting RNA polymerase III through ERK1/2
Rationale: An imbalance between protein synthesis and degradation is one of the mechanisms of cardiac hypertrophy. Increased transcription in cardiomyocytes can lead to excessive protein synthesis and cardiac hypertrophy. Maf1 is an RNA polymerase III (RNA pol III) inhibitor that plays a pivotal rol...
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| Pubblicato in: | Theranostics |
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| Autori principali: | , , , , , , , , , , , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Ivyspring International Publisher
2019
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6831308/ https://ncbi.nlm.nih.gov/pubmed/31695767 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7150/thno.33006 |
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