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Replication Stress Response Links RAD52 to Protecting Common Fragile Sites
Rad52 in yeast is a key player in homologous recombination (HR), but mammalian RAD52 is dispensable for HR as shown by the lack of a strong HR phenotype in RAD52-deficient cells and in RAD52 knockout mice. RAD52 function in mammalian cells first emerged with the discovery of its important backup rol...
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| Publicado no: | Cancers (Basel) |
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| Autor principal: | |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
MDPI
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6826974/ https://ncbi.nlm.nih.gov/pubmed/31569559 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/cancers11101467 |
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