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Traumatic Stress Produces Delayed Alterations of Synaptic Plasticity in Basolateral Amygdala
Acute traumatic event exposure is a direct cause of post-traumatic stress disorder (PTSD). Amygdala is suggested to be associated with the development of PTSD. In our previous findings, different activation patterns of GABAergic neurons and glutamatergic neurons in early or late stages after stress...
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| I publikationen: | Front Psychol |
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| Huvudupphovsmän: | , , , , , , , , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
Frontiers Media S.A.
2019
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6824323/ https://ncbi.nlm.nih.gov/pubmed/31708835 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fpsyg.2019.02394 |
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