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Traumatic Stress Produces Delayed Alterations of Synaptic Plasticity in Basolateral Amygdala
Acute traumatic event exposure is a direct cause of post-traumatic stress disorder (PTSD). Amygdala is suggested to be associated with the development of PTSD. In our previous findings, different activation patterns of GABAergic neurons and glutamatergic neurons in early or late stages after stress...
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| 出版年: | Front Psychol |
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| 主要な著者: | , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Frontiers Media S.A.
2019
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6824323/ https://ncbi.nlm.nih.gov/pubmed/31708835 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fpsyg.2019.02394 |
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