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Traumatic Stress Produces Delayed Alterations of Synaptic Plasticity in Basolateral Amygdala
Acute traumatic event exposure is a direct cause of post-traumatic stress disorder (PTSD). Amygdala is suggested to be associated with the development of PTSD. In our previous findings, different activation patterns of GABAergic neurons and glutamatergic neurons in early or late stages after stress...
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| Опубликовано в: : | Front Psychol |
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| Главные авторы: | , , , , , , , , |
| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
Frontiers Media S.A.
2019
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6824323/ https://ncbi.nlm.nih.gov/pubmed/31708835 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fpsyg.2019.02394 |
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