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Differential Mechanism of ATP Production Occurs in Response to Succinylacetone in Colon Cancer Cells
Our aim was to verify the potential ability of succinylacetone (SA) to inhibit mitochondrial function, thereby suppressing cancer cell proliferation. SA treatment caused apoptosis in HCT116 and HT29 cells, but not in SW480 cells, with mitochondria playing a key role. We checked for dysfunctional mit...
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| Veröffentlicht in: | Molecules |
|---|---|
| Hauptverfasser: | , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
MDPI
2019
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6803967/ https://ncbi.nlm.nih.gov/pubmed/31623369 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/molecules24193575 |
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