Soluble RAGE attenuates AngII-induced endothelial hyperpermeability by disrupting HMGB1-mediated crosstalk between AT1R and RAGE

Increased endothelial permeability, one of the earliest signs of endothelial dysfunction, is associated with the development of cardiovascular diseases such as hypertension and atherosclerosis. Recent studies suggest that the receptor for advanced glycation end products (RAGE) regulates endothelial...

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Detalhes bibliográficos
Publicado no:Exp Mol Med
Main Authors: Jeong, Jisu, Lee, Jiye, Lim, Juyeon, Cho, Soyoung, An, Soyoung, Lee, Myungeun, Yoon, Nara, Seo, Miran, Lim, Soyeon, Park, Sungha
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2019
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6802637/
https://ncbi.nlm.nih.gov/pubmed/31562296
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s12276-019-0312-5
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