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TAFI deficiency causes maladaptive vascular remodeling after hemophilic joint bleeding
Excessive vascular remodeling is characteristic of hemophilic arthropathy (HA) and may contribute to joint bleeding and the progression of HA. Mechanisms for pathological vascular remodeling after hemophilic joint bleeding are unknown. In hemophilia, activation of thrombin-activatable fibrinolysis i...
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| Udgivet i: | JCI Insight |
|---|---|
| Main Authors: | , , , , , , , , , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
American Society for Clinical Investigation
2019
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6795396/ https://ncbi.nlm.nih.gov/pubmed/31465300 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.128379 |
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