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Lambert-Eaton Antibodies Inhibit Ca(2+) Currents But Paradoxically Increase Exocytosis during Stimulus Trains in Bovine Adrenal Chromaffin Cells
Lambert-Eaton myasthenic syndrome (LEMS) is an autoimmune disease that affects neurotransmitter release at peripheral synapses. LEMS antibodies inhibit Ca(2+) currents in excitable cells, but it is not known whether there are additional effects on stimulus-secretion coupling. The effect of LEMS anti...
Tallennettuna:
| Julkaisussa: | J Neurosci |
|---|---|
| Päätekijät: | , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Society for Neuroscience
1999
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6782246/ https://ncbi.nlm.nih.gov/pubmed/10212298 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.19-09-03384.1999 |
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