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Ataxin2 functions via CrebA to mediate Huntingtin toxicity in circadian clock neurons

Disrupted circadian rhythms is a prominent and early feature of neurodegenerative diseases including Huntington’s disease (HD). In HD patients and animal models, striatal and hypothalamic neurons expressing molecular circadian clocks are targets of mutant Huntingtin (mHtt) pathogenicity. Yet how mHt...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:PLoS Genet
Päätekijät: Xu, Fangke, Kula-Eversole, Elzbieta, Iwanaszko, Marta, Lim, Chunghun, Allada, Ravi
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Public Library of Science 2019
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6782096/
https://ncbi.nlm.nih.gov/pubmed/31593562
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pgen.1008356
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