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The Lurcher Mutation Identifies δ2 as an AMPA/Kainate Receptor-Like Channel That Is Potentiated by Ca(2+)

Neurodegeneration in Lurcher (Lc) mice results from constitutive activation of δ2, a subunit of ionotropic glutamate receptors (GluRs) with unknown natural ligands and channel properties. Homo-oligomeric channels of GluR-δ2 with the Lurcher mutation (GluR-δ2(Lc)) expressed in human embryonic kidney...

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Detaylı Bibliyografya
Yayımlandı:J Neurosci
Asıl Yazarlar: Wollmuth, Lonnie P., Kuner, Thomas, Jatzke, Claudia, Seeburg, Peter H., Heintz, Nathaniel, Zuo, Jian
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Society for Neuroscience 2000
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6772614/
https://ncbi.nlm.nih.gov/pubmed/10934245
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.20-16-05973.2000
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