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Phosphorylation State of Ser(165) in α-Tubulin is a Toggle Switch That Controls Proliferating Human Breast Tumors
Engineered overexpression of protein kinase Cα (PKCα) is known to phosphorylate Ser(165) in α-tubulin resulting in stimulated microtubule dynamics and cell motility, and activation of an epithelial-mesenchymal transition (EMT) in non-transformed human breast cells. Here it is shown that endogenous p...
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| Pubblicato in: | Cell Signal |
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| Autori principali: | , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2018
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6765385/ https://ncbi.nlm.nih.gov/pubmed/30176291 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cellsig.2018.08.021 |
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