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M Channel KCNQ2 Subunits Are Localized to Key Sites for Control of Neuronal Network Oscillations and Synchronization in Mouse Brain
Mutations in the potassium channel subunit KCNQ2 lead to benign familial neonatal convulsions, a dominantly inherited form of generalized epilepsy. In heterologous cells, KCNQ2 expression yields voltage-gated potassium channels that activate slowly (τ, ∼0.1 sec) at subthreshold membrane potentials....
Tallennettuna:
| Julkaisussa: | J Neurosci |
|---|---|
| Päätekijät: | , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Society for Neuroscience
2001
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6763050/ https://ncbi.nlm.nih.gov/pubmed/11739564 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.21-24-09529.2001 |
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