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Severe Impairment of NMDA Receptor Function in Mice Carrying Targeted Point Mutations in the Glycine Binding Site Results in Drug-Resistant Nonhabituating Hyperactivity

NMDA receptor hypofunction has been implicated in the pathophysiology of schizophrenia, and pharmacological and genetic approaches have been used to model such dysfunction. We previously have described two mouse lines carrying point mutations in the NMDA receptor glycine binding site,Grin1(D481N) an...

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הוצא לאור ב:	J Neurosci
Main Authors:	Ballard, Theresa M., Pauly-Evers, Meike, Higgins, Guy A., Ouagazzal, Abdel-Mouttalib, Mutel, Vincent, Borroni, Edilio, Kemp, John A., Bluethmann, Horst, Kew, James N. C.
פורמט:	Artigo
שפה:	Inglês
יצא לאור:	Society for Neuroscience 2002
נושאים:	ARTICLE
גישה מקוונת:	https://ncbi.nlm.nih.gov/pmc/articles/PMC6758156/ https://ncbi.nlm.nih.gov/pubmed/12151550 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.22-15-06713.2002
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Severe Impairment of NMDA Receptor Function in Mice Carrying Targeted Point Mutations in the Glycine Binding Site Results in Drug-Resistant Nonhabituating Hyperactivity

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