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CED-4 CARD domain residues can modulate non-apoptotic neuronal regeneration functions independently from apoptosis
A major challenge in regenerative medicine is the repair of injured neurons. Regeneration of laser-cut C. elegans neurons requires early action of core apoptosis activator CED-4/Apaf1 and CED-3/caspase. While testing models for CED-4 as a candidate calcium-sensitive activator of repair, we unexpecte...
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| Publicado no: | Sci Rep |
|---|---|
| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group UK
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6746752/ https://ncbi.nlm.nih.gov/pubmed/31527664 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-019-49633-9 |
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