Apoptosis Induced by p75NTR Overexpression Requires Jun Kinase-Dependent Phosphorylation of Bad

The p75 neurotrophin receptor (p75NTR), a member of the tumor necrosis factor receptor superfamily, facilitates apoptosis during development and after injury to the CNS. The signaling cascades activated by p75NTR that result in apoptosis remain poorly understood. In this study, we show that overexpr...

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Опубликовано в: :J Neurosci
Главные авторы: Bhakar, Asha L., Howell, Jenny L., Paul, Christine E., Salehi, Amir H., Becker, Esther B. E., Said, Farid, Bonni, Azad, Barker, Philip A.
Формат: Artigo
Язык:Inglês
Опубликовано: Society for Neuroscience 2003
Предметы:
Cellular/Molecular
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC6740508/
https://ncbi.nlm.nih.gov/pubmed/14673001
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.23-36-11373.2003
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