TRAF3IP2 mediates TWEAK/TWEAKR-induced pro-fibrotic responses in cultured cardiac fibroblasts and the heart

Persistent inflammation promotes development and progression of heart failure (HF). TWEAK (TNF-Related WEAK Inducer Of Apoptosis), a NF-κB- and/or AP-1-responsive proinflammatory cytokine that signals via TWEAK receptor (TWEAKR), is expressed at high levels in human and preclinical models of HF. Sin...

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Detalhes bibliográficos
Publicado no:J Mol Cell Cardiol
Main Authors: Das, Nitin A., Carpenter, Andrea J., Yoshida, Tadashi, Kumar, Senthil A., Gautam, Sandeep, Mostany, Ricardo, Izadpanah, Reza, Kumar, Ashok, Mummidi, Srinivas, Siebenlist, Ulrich, Chandrasekar, Bysani
Formato: Artigo
Idioma:Inglês
Publicado em: 2018
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6732793/
https://ncbi.nlm.nih.gov/pubmed/29981796
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2018.07.003
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