CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction

Hyperglycemia-induced endothelial dysfunction is characterized by enhanced inflammatory cytokine and adhesion molecule expression, and endothelial-monocyte adhesion. The adapter molecule CIKS (connection to IKK and SAPK/JNK; also known as Act1 or TRAF3IP2) mediates NF-κB and AP-1 activation, and pla...

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Hlavní autoři: Venkatesan, Balachandar, Valente, Anthony J., Das, Nitin A., Carpenter, Andrea J., Yoshida, Tadashi, Delafontaine, Jean-Luc, Siebenlist, Ulrich, Chandrasekar, Bysani
Médium: Artigo
Jazyk:Inglês
Vydáno: 2012
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3606809/
https://ncbi.nlm.nih.gov/pubmed/23085260
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cellsig.2012.10.009
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