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Sequential Release of GABA by Exocytosis and Reversed Uptake Leads to Neuronal Swelling in Simulated Ischemia of Hippocampal Slices

GABA release during cerebral energy deprivation (produced by anoxia or ischemia) has been suggested either to be neuroprotective, because GABA will hyperpolarize neurons and reduce release of excitotoxic glutamate, or to be neurotoxic, because activation of GABA(A) receptors facilitates Cl(–) entry...

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Bibliografske podrobnosti
izdano v:J Neurosci
Main Authors: Allen, Nicola J., Rossi, David J., Attwell, David
Format: Artigo
Jezik:Inglês
Izdano: Society for Neuroscience 2004
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC6729351/
https://ncbi.nlm.nih.gov/pubmed/15084665
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.5539-03.2004
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