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Sequential Release of GABA by Exocytosis and Reversed Uptake Leads to Neuronal Swelling in Simulated Ischemia of Hippocampal Slices

GABA release during cerebral energy deprivation (produced by anoxia or ischemia) has been suggested either to be neuroprotective, because GABA will hyperpolarize neurons and reduce release of excitotoxic glutamate, or to be neurotoxic, because activation of GABA(A) receptors facilitates Cl(–) entry...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
में प्रकाशित:J Neurosci
मुख्य लेखकों: Allen, Nicola J., Rossi, David J., Attwell, David
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: Society for Neuroscience 2004
विषय:
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC6729351/
https://ncbi.nlm.nih.gov/pubmed/15084665
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.5539-03.2004
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