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Transforming Growth Factor β1 Promotes Cell Cycle Exit through the Cyclin-Dependent Kinase Inhibitor p21 in the Developing Cerebral Cortex
During cortical neurogenesis, cell proliferation and cell cycle exit are carefully regulated to ensure that the appropriate numbers of cells are produced. The antiproliferative agent transforming growth factor β1 (TGFβ1) and its receptors are endogenously expressed in proliferative zones of the deve...
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| Published in: | J Neurosci |
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| Main Authors: | , |
| Format: | Artigo |
| Language: | Inglês |
| Published: |
Society for Neuroscience
2005
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| Subjects: | |
| Online Access: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6725510/ https://ncbi.nlm.nih.gov/pubmed/16177030 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1876-05.2005 |
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