Nitric oxide maintains endothelial redox homeostasis through PKM2 inhibition

Decreased nitric oxide (NO) bioavailability and oxidative stress are hallmarks of endothelial dysfunction and cardiovascular diseases. Although numerous proteins are S‐nitrosated, whether and how changes in protein S‐nitrosation influence endothelial function under pathophysiological conditions rema...

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Detalles Bibliográficos
Publicado en:EMBO J
Main Authors: Siragusa, Mauro, Thöle, Janina, Bibli, Sofia‐Iris, Luck, Bert, Loot, Annemarieke E, de Silva, Kevin, Wittig, Ilka, Heidler, Juliana, Stingl, Heike, Randriamboavonjy, Voahanginirina, Kohlstedt, Karin, Brüne, Bernhard, Weigert, Andreas, Fisslthaler, Beate, Fleming, Ingrid
Formato: Artigo
Idioma:Inglês
Publicado: John Wiley and Sons Inc. 2019
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Articles
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC6717893/
https://ncbi.nlm.nih.gov/pubmed/31328803
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/embj.2018100938
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