Nitric oxide maintains endothelial redox homeostasis through PKM2 inhibition

Decreased nitric oxide (NO) bioavailability and oxidative stress are hallmarks of endothelial dysfunction and cardiovascular diseases. Although numerous proteins are S‐nitrosated, whether and how changes in protein S‐nitrosation influence endothelial function under pathophysiological conditions rema...

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Enregistré dans:
Détails bibliographiques
Publié dans:EMBO J
Auteurs principaux: Siragusa, Mauro, Thöle, Janina, Bibli, Sofia‐Iris, Luck, Bert, Loot, Annemarieke E, de Silva, Kevin, Wittig, Ilka, Heidler, Juliana, Stingl, Heike, Randriamboavonjy, Voahanginirina, Kohlstedt, Karin, Brüne, Bernhard, Weigert, Andreas, Fisslthaler, Beate, Fleming, Ingrid
Format: Artigo
Langue:Inglês
Publié: John Wiley and Sons Inc. 2019
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Articles
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC6717893/
https://ncbi.nlm.nih.gov/pubmed/31328803
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/embj.2018100938
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