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New thiazolidinones reduce iron overload in mouse models of hereditary hemochromatosis and β-thalassemia
Genetic iron-overload disorders, mainly hereditary hemochromatosis and untransfused β-thalassemia, affect a large population worldwide. The primary etiology of iron overload in these diseases is insufficient production of hepcidin by the liver, leading to excessive intestinal iron absorption and iro...
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| Publicado no: | Haematologica |
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| Main Authors: | , , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Ferrata Storti Foundation
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6717595/ https://ncbi.nlm.nih.gov/pubmed/30792208 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3324/haematol.2018.209874 |
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