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CD44 inhibits α-SMA gene expression via a novel G-actin/MRTF-mediated pathway that intersects with TGFβR/p38MAPK signaling in murine skin fibroblasts

Well-regulated differentiation of fibroblasts into myofibroblasts (MF) is critical for skin wound healing. Neoexpression of α-smooth muscle actin (α-SMA), an established marker for MF differentiation, is driven by TGFβ receptor (TGFβR)–mediated signaling. Hyaluronan (HA) and its receptor CD44 may al...

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Bibliografske podrobnosti
izdano v:J Biol Chem
Main Authors: Wang, Yan, Mack, Judith A., Maytin, Edward V.
Format: Artigo
Jezik:Inglês
Izdano: American Society for Biochemistry and Molecular Biology 2019
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC6709637/
https://ncbi.nlm.nih.gov/pubmed/31285260
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.RA119.007834
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