Endogenous AT1 Receptor–Protein Kinase C Activity in the Hypothalamus Augments Glutamatergic Input and Sympathetic Outflow in Hypertension

Lignende værker

• Src Kinases Regulate Glutamatergic Input to Hypothalamic Presympathetic Neurons and Sympathetic Outflow in Hypertension
  af: Qiao, Xin, et al.
  Udgivet: (2016)
• Protein Kinase CK2 Increases Glutamatergic Input in the Hypothalamus and Sympathetic Vasomotor Tone in Hypertension
  af: Ye, Zeng-You, et al.
  Udgivet: (2011)
• α2δ‐1 couples to NMDA receptors in the hypothalamus to sustain sympathetic vasomotor activity in hypertension
  af: Ma, Huijie, et al.
  Udgivet: (2018)
• Impaired Hypothalamic Regulation of Sympathetic Outflow in Primary Hypertension
  af: Zhou, Jing-Jing, et al.
  Udgivet: (2018)
• Endogenous casein kinase‐1 modulates NMDA receptor activity of hypothalamic presympathetic neurons and sympathetic outflow in hypertension
  af: Li, De‐Pei, et al.
  Udgivet: (2015)