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p35/p25 Is Not Essential for Tau and Cytoskeletal Pathology or Neuronal Loss in Niemann–Pick Type C Disease

Hyperactivation of the cyclin-dependent kinase 5 (cdk5), triggered by proteolytic conversion of its neuronal activator, p35, to a more potent byproduct, p25, has been implicated in Alzheimer's disease (AD), amyotrophic lateral sclerosis, and Niemann–Pick type C disease (NPC). This mechanism is...

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Dades bibliogràfiques
Publicat a:J Neurosci
Autors principals: Hallows, Janice L., Iosif, Robert E., Biasell, Rebecca D., Vincent, Inez
Format: Artigo
Idioma:Inglês
Publicat: Society for Neuroscience 2006
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC6675168/
https://ncbi.nlm.nih.gov/pubmed/16525053
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4834-05.2006
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