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Pathway-Specific Bidirectional Regulation of Ca(2+)/Calmodulin-Dependent Protein Kinase II at Spinal Nociceptive Synapses after Acute Noxious Stimulation

An intensely painful stimulus may lead to hyperalgesia, the enhanced sensation of subsequent painful stimuli. This is commonly believed to involve facilitated transmission of sensory signals in the spinal cord, possibly by a long-term potentiation-like mechanism. However, plasticity of identified sy...

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Detalles Bibliográficos
Publicado en:J Neurosci
Main Authors: Larsson, Max, Broman, Jonas
Formato: Artigo
Idioma:Inglês
Publicado: Society for Neuroscience 2006
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC6674005/
https://ncbi.nlm.nih.gov/pubmed/16624940
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0352-06.2006
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