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Pathway-Specific Bidirectional Regulation of Ca(2+)/Calmodulin-Dependent Protein Kinase II at Spinal Nociceptive Synapses after Acute Noxious Stimulation
An intensely painful stimulus may lead to hyperalgesia, the enhanced sensation of subsequent painful stimuli. This is commonly believed to involve facilitated transmission of sensory signals in the spinal cord, possibly by a long-term potentiation-like mechanism. However, plasticity of identified sy...
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| Pubblicato in: | J Neurosci |
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| Autori principali: | , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Society for Neuroscience
2006
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6674005/ https://ncbi.nlm.nih.gov/pubmed/16624940 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0352-06.2006 |
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