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Pathway-Specific Bidirectional Regulation of Ca(2+)/Calmodulin-Dependent Protein Kinase II at Spinal Nociceptive Synapses after Acute Noxious Stimulation

An intensely painful stimulus may lead to hyperalgesia, the enhanced sensation of subsequent painful stimuli. This is commonly believed to involve facilitated transmission of sensory signals in the spinal cord, possibly by a long-term potentiation-like mechanism. However, plasticity of identified sy...

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Vydáno v:J Neurosci
Hlavní autoři: Larsson, Max, Broman, Jonas
Médium: Artigo
Jazyk:Inglês
Vydáno: Society for Neuroscience 2006
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6674005/
https://ncbi.nlm.nih.gov/pubmed/16624940
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0352-06.2006
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