Free Radical Production in CA1 Neurons Induces MIP-1α Expression, Microglia Recruitment, and Delayed Neuronal Death after Transient Forebrain Ischemia

Several studies report microglial accumulation and activation in the CA1 area in response to transient forebrain ischemia (TFI). Here we examine the possibility that free radicals and chemokines mediate the transient activation of microglia. Free radicals are produced primarily in CA1 pyramidal neur...

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Detaylı Bibliyografya
Yayımlandı:J Neurosci
Asıl Yazarlar: Wang, Hyo Kyun, Park, Ui Jin, Kim, Soo Yoon, Lee, Jin Hwan, Kim, Seung U., Gwag, Byoung Joo, Lee, Yong Beom
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Society for Neuroscience 2008
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Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6671544/
https://ncbi.nlm.nih.gov/pubmed/18272692
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4973-07.2008
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