Free Radical Production in CA1 Neurons Induces MIP-1α Expression, Microglia Recruitment, and Delayed Neuronal Death after Transient Forebrain Ischemia

Several studies report microglial accumulation and activation in the CA1 area in response to transient forebrain ischemia (TFI). Here we examine the possibility that free radicals and chemokines mediate the transient activation of microglia. Free radicals are produced primarily in CA1 pyramidal neur...

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Xehetasun bibliografikoak
Argitaratua izan da:J Neurosci
Egile Nagusiak: Wang, Hyo Kyun, Park, Ui Jin, Kim, Soo Yoon, Lee, Jin Hwan, Kim, Seung U., Gwag, Byoung Joo, Lee, Yong Beom
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Society for Neuroscience 2008
Gaiak:
Articles
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC6671544/
https://ncbi.nlm.nih.gov/pubmed/18272692
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4973-07.2008
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