Free Radical Production in CA1 Neurons Induces MIP-1α Expression, Microglia Recruitment, and Delayed Neuronal Death after Transient Forebrain Ischemia

Several studies report microglial accumulation and activation in the CA1 area in response to transient forebrain ischemia (TFI). Here we examine the possibility that free radicals and chemokines mediate the transient activation of microglia. Free radicals are produced primarily in CA1 pyramidal neur...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Wang, Hyo Kyun, Park, Ui Jin, Kim, Soo Yoon, Lee, Jin Hwan, Kim, Seung U., Gwag, Byoung Joo, Lee, Yong Beom
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2008
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6671544/
https://ncbi.nlm.nih.gov/pubmed/18272692
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4973-07.2008
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