Glycinergic and GABA(A)-Mediated Inhibition of Somatic Motoneurons Does Not Mediate Rapid Eye Movement Sleep Motor Atonia

Ítems similars

• Are All Motoneurons Created Equal in the Eyes of REM Sleep and the Mechanisms of Muscle Atonia?: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
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• Confirmation of the Consensus that Glycinergic Postsynaptic Inhibition is Responsible for the Atonia of REM Sleep: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
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• What Causes Muscle Atonia in REM?: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
  per: Berger, Albert J.
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• Adventures and Tribulations in the Search for the Mechanisms of the Atonia of REM Sleep: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
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• Glycine-Mediated Postsynaptic Inhibition is Responsible for REM Sleep Atonia: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
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  Publicat: (2008)