Glycine-Mediated Postsynaptic Inhibition is Responsible for REM Sleep Atonia: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.

Ítems similars

• Confirmation of the Consensus that Glycinergic Postsynaptic Inhibition is Responsible for the Atonia of REM Sleep: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
  per: Chase, Michael H.
  Publicat: (2008)
• What Causes Muscle Atonia in REM?: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
  per: Berger, Albert J.
  Publicat: (2008)
• Adventures and Tribulations in the Search for the Mechanisms of the Atonia of REM Sleep: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
  per: Kubin, Leszek
  Publicat: (2008)
• Are All Motoneurons Created Equal in the Eyes of REM Sleep and the Mechanisms of Muscle Atonia?: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
  per: Funk, Gregory D.
  Publicat: (2008)
• Glycinergic and GABA(A)-Mediated Inhibition of Somatic Motoneurons Does Not Mediate Rapid Eye Movement Sleep Motor Atonia
  per: Brooks, Patricia L., et al.
  Publicat: (2008)