All-trans retinoic acid attenuates isoproterenol-induced cardiac dysfunction through Crabp1 to dampen CaMKII activation

Inhibiting Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) over activation can decrease detrimental cardiac remodeling that leads to dilated cardiomyopathy, cell death, and heart failure. We previously showed that cellular retinoic acid binding protein 1 (Crabp1) knockout mice (CKO) exhibited...

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Bibliografiska uppgifter
I publikationen:Eur J Pharmacol
Huvudupphovsmän: Park, Sung Wook, Nhieu, Jennifer, Lin, Yi-Wei, Wei, Li-Na
Materialtyp: Artigo
Språk:Inglês
Publicerad: 2019
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Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC6667285/
https://ncbi.nlm.nih.gov/pubmed/31238067
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ejphar.2019.172485
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