α9 Integrin Promotes Neurite Outgrowth on Tenascin-C and Enhances Sensory Axon Regeneration

Damaged CNS axons are prevented from regenerating by an environment containing many inhibitory factors. They also lack an integrin that interacts with tenascin-C, the main extracellular matrix glycoprotein of the CNS, which is upregulated after injury. The α9β1 integrin heterodimer is a receptor for...

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Bibliografiset tiedot
Julkaisussa:J Neurosci
Päätekijät: Andrews, Melissa R., Czvitkovich, Stefan, Dassie, Elisa, Vogelaar, Christina F., Faissner, Andreas, Blits, Bas, Gage, Fred H., ffrench-Constant, Charles, Fawcett, James W.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Society for Neuroscience 2009
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Articles
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6665849/
https://ncbi.nlm.nih.gov/pubmed/19403822
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0759-09.2009
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