טוען...
α9 Integrin Promotes Neurite Outgrowth on Tenascin-C and Enhances Sensory Axon Regeneration
Damaged CNS axons are prevented from regenerating by an environment containing many inhibitory factors. They also lack an integrin that interacts with tenascin-C, the main extracellular matrix glycoprotein of the CNS, which is upregulated after injury. The α9β1 integrin heterodimer is a receptor for...
שמור ב:
| הוצא לאור ב: | J Neurosci |
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| Main Authors: | , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Society for Neuroscience
2009
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6665849/ https://ncbi.nlm.nih.gov/pubmed/19403822 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0759-09.2009 |
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