The Absence of Endothelial Sodium Channel α (αENaC) Reduces Renal Ischemia/Reperfusion Injury

The epithelial sodium channel (ENaC) has a key role in modulating endothelial cell stiffness and this in turn regulates nitric oxide (NO) synthesis. The physiological relevance of endothelial ENaC in pathological conditions where reduced NO bioavailability plays an essential role remains largely une...

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Detalles Bibliográficos
Publicado en:Int J Mol Sci
Main Authors: Tarjus, Antoine, González-Rivas, Cecilia, Amador-Martínez, Isabel, Bonnard, Benjamin, López-Marure, Rebeca, Jaisser, Frédéric, Barrera-Chimal, Jonatan
Formato: Artigo
Idioma:Inglês
Publicado: MDPI 2019
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Article
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC6651193/
https://ncbi.nlm.nih.gov/pubmed/31252520
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/ijms20133132
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