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AAV2/9-mediated overexpression of MIF inhibits SOD1 misfolding, delays disease onset, and extends survival in mouse models of ALS

Mutations in superoxide dismutase (SOD1) cause amyotrophic lateral sclerosis (ALS), a neurodegenerative disease characterized by the loss of upper and lower motor neurons. Transgenic mice that overexpress mutant SOD1 develop paralysis and accumulate misfolded SOD1 onto the cytoplasmic faces of intra...

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Foilsithe in:Proc Natl Acad Sci U S A
Main Authors: Leyton-Jaimes, Marcel F., Kahn, Joy, Israelson, Adrian
Formáid: Artigo
Teanga:Inglês
Foilsithe: National Academy of Sciences 2019
Ábhair:
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC6642395/
https://ncbi.nlm.nih.gov/pubmed/31262807
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1904665116
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