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Deletion of translin (Tsn) induces robust adiposity and hepatic steatosis without impairing glucose tolerance

OBJECTIVE: Translin knockout (KO) mice display robust adiposity. Recent studies indicate that translin and its partner protein, trax, regulate the microRNA and ATM kinase signaling pathways, both of which have been implicated in regulating metabolism. In the course of characterizing the metabolic pr...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Int J Obes (Lond)
Päätekijät: Shah, Aparna P., Johnson, Miranda D., Fu, Xiuping, Boersma, Gretha J., Shah, Madhura, Wolfgang, Michael J., Tamashiro, Kellie L., Baraban, Jay M.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2019
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6629527/
https://ncbi.nlm.nih.gov/pubmed/30647452
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41366-018-0315-7
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