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Cannabinoid receptor 2 deficiency exacerbates inflammation and neutrophil recruitment
Cannabinoid receptor (CB)(2) is an immune cell–localized GPCR that has been hypothesized to regulate the magnitude of inflammatory responses. However, there is currently no consensus as to the mechanism by which CB(2) mediates its anti-inflammatory effects in vivo. To address this question, we emplo...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | FASEB J |
|---|---|
| Κύριοι συγγραφείς: | , , , , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
Federation of American Societies for Experimental Biology
2019
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6629158/ https://ncbi.nlm.nih.gov/pubmed/30799631 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.201802524R |
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