Alternative Splicing of the TRPC3 Ion Channel Calmodulin/IP(3) Receptor-Binding Domain in the Hindbrain Enhances Cation Flux

Canonical transient receptor potential (TRPC3) nonselective cation channels are effectors of G-protein-coupled receptors (GPCRs), activated via phospholipase C–diacylglycerol signaling. In cerebellar Purkinje cells, TRPC3 channels cause the metabotropic glutamate receptor (mGluR)-mediated slow EPSC...

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Bibliografiset tiedot
Julkaisussa:J Neurosci
Päätekijät: Kim, Youngsoo, Wong, Ann Chi Yan, Power, John M., Tadros, Sherif F., Klugmann, Matthias, Moorhouse, Andrew J., Bertrand, Paul P., Housley, Gary D.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Society for Neuroscience 2012
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Articles
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6621195/
https://ncbi.nlm.nih.gov/pubmed/22895723
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.6446-11.2012
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