Distinct MRAP domains are required for MC2R interaction and promotion of receptor trafficking

Lignende værker

• MRAP and MRAP2 are bidirectional regulators of the melanocortin receptor family
  af: Chan, Li F., et al.
  Udgivet: (2009)
• The majority of ACTH receptor (MC2R) mutations found in Familial Glucocorticoid Deficiency type 1 lead to defective trafficking of the receptor to the cell surface
  af: TT, Chung, et al.
  Udgivet: (2008)
• Opposite Effects of the Melanocortin-2 (MC2) Receptor Accessory Protein MRAP on MC2 and MC5 Receptor Dimerization and Trafficking
  af: Sebag, Julien A., et al.
  Udgivet: (2009)
• Isolated Addison's disease is unlikely to be caused by mutations in MC2R, MRAP or STAR, three genes responsible for familial glucocorticoid deficiency
  af: Dias, R P, et al.
  Udgivet: (2010)
• hMRAPα, but Not hMRAP2, Enhances hMC4R Constitutive Activity in HEK293 Cells and This Is Not Dependent on hMRAPα Induced Changes in hMC4R Complex N-linked Glycosylation
  af: Kay, Emma I., et al.
  Udgivet: (2015)